Preliminary scientific studies indicate conolidine could inhibit certain ion channels, minimizing neuronal excitability and limiting agony alerts. This mechanism is especially relevant in neuropathic soreness, where abnormal signaling leads to persistent soreness. Furthermore, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to pain notion. Computationa... https://elliottk268spl9.get-blogging.com/profile