Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Then again, our ability to rescue SERT purpose in KI midbrain synaptoneurosomes because of the inhibition of FAK implies elevated FAK signaling downstream in the Pro32Pro33 mutant, as verified by amplified pFAK localization in 5-HT https://alphonsec108gsc9.goabroadblog.com/profile